Donguibogam on Blood Stasis Tinnitus: Restoring Ear Health by Improving Blood Circulation

The concept of blood stasis in the Donguibogam corresponds in modern medicine to conditions such as microvessel dysfunction, increased blood viscosity, and vascular endothelial cell damage. Inside the inner ear, particularly within the cochlea responsible for hearing, microscopic capillaries form a dense network. These microvessels supply oxygen and nutrients to auditory hair cells and nerves, which are among the most sensitive tissues in the body. Similar vascular issues occur frequently in individuals with chronic conditions such as hypertension, diabetes, and hyperlipidemia. Hypertension places continuous high pressure on blood vessels, hardening and damaging the vascular wall over time. Diabetes, through poorly managed blood glucose, damages microvessels and increases blood viscosity, impeding circulation. Narrowed and damaged vessels compromise blood flow to the inner ear and can even lead to small thrombi that obstruct vessels completely. Auditory hair cells are highly sensitive to oxygen deprivation, beginning to sustain damage within minutes if oxygen delivery is interrupted. When circulation is compromised, these hair cells malfunction or degenerate, sending abnormal electrical impulses to the brain that are perceived as tinnitus. This is similar to static produced when a broadcast signal becomes unstable. In addition, chronic inflammation and oxidative stress contribute significantly to tinnitus. Vascular endothelial cell damage triggers an inflammatory response, which further impairs circulation in a continuous cycle. Tinnitus is therefore not simply a localized ear issue, but a systemic warning sign that circulation and metabolism require timely evaluation and care. Leaving tinnitus unaddressed may coincide with the worsening of underlying conditions such as hypertension or diabetes, and it warrants careful attention.